Sudden Infant Death Syndrome (SIDS) has many different etiologies. Inability to use fatty acids (FA) for energy when the body is low on glucose, which is especially important for the heart and for generating heat in cold environments, has been shown in 5-15%of cases. Most of the time, the reason the infant cannot use FA is because of a defect in an enzyme involved in breaking down FA. CD36 delivers FA to cells, and the expression of CD36 has been found to vary a lot from person to person and cell to cell. We think that a deficiency in delivery of FA to cells will mimic an enzyme defect, in that the infant will not be able to breakdown FA for energy because they are in short supply. Thus, we think differences in CD36 expression may be an etiology for SIDS.